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Evidences from human studies. Amino acid neurotransmitters assessed by proton magnetic resonance spectroscopy: There is a circadian peak of MI onset in the morning hours, and people may drink more alcohol in the evening and night. The density and spatial distribution of GABAergic neurons, labelled using calcium binding proteins, in the anterior cingulate cortex in major depressive disorder, bipolar disorder, and schizophrenia.

Advantages and disadvantages of combination treatment with antipsychotics. Cytokine production and treatment response in major depressive disorder. Lifestyle determinants of high-density lipoprotein cholesterol: Pharmacol Biochem Behav ; A longitudinal study of hippocampal volume, cortisol levels, and cognition in older depressed subjects.


The association was stronger for liquor than for beer or wine. Dose-related effects of red wine and alcohol on hemodynamics, sympathetic nerve activity, and arterial diameter. Prog Neuropsychopharmacol Biol Psychiat.

J Neurol Neurosurg Psychiatry. Petty F, Schiesser MA. A meta-analysis of comparative studies. Am J Psychiatry ; Agomelatine in the treatment of major depressive disorder: Acute alcohol consumption is associated with increased interatrial electromechanical delay in healthy men.


Author manuscript; available in PMC Mar 1. J Neuropsychiatry Clin Neurosci. Therefore, the susceptible pool would be depleted in the 2—24 hours after the hypothesized hazard of 1 hour after alcohol consumption; this may at least partially explain our finding that alcohol intake is associated with lower MI risk within 24 hours.

The clinical relevance of antidepressant interaction with neurotransmitter transporters and receptors. Remodeling of hippocampal spine synapses in the rat learned helplessness model of depression.

GABA in normal subjects and patients with depression, schizophrenia, mania, and anorexia nervosa.

To estimate the length of time from alcohol consumption to MI onset, we calculated RRs for various hypothesized windows of its physiologic effect. Habitual moderate alcohol consumption is associated with a lower risk of acute myocardial infarction MI whereas heavy binge drinking is associated with higher cardiovascular risk.


The GABAergic deficit hypothesis of major depressive disorder. Glucocorticoids, depression, and mood disorders: Current understanding of the neurobiology of major depressive disorder. Relative risk of acute myocardial infarction in the hour after consumption of beer, wine or liquor.

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We identified the time, place and quality of the acute MI pain and other symptoms. Depression, stress and immunological activation: Subtypespecific alterations of gamma-aminobutyric acid and glutamate in patients with major depression.

Reduced hippocampal volume in unmedicated, remitted patients with major depression versus control subjects. A neurotrophic model for stress-related mood disorders.